About Dr. Mohammed Al-Bayati
|Use of EJ Scovills lung tissue as a negative control in the case of Averial Buie|
|Comparison of EJ Scovill and Destiny Jacobo cases in the journal Medical Veritas|
|Hisopathological report in the journal Medical Veritas|
October 21, 2006
|Histopathological report (analysis of prepared microscope slides).|
|Analysis of the Causes of Eliza Jane Scovills Death in the journal Medical Veritas|
December 9, 2005
|Comparative Analysis of the Autopsy Reports of Destiny Jacobo and Eliza Jane Scovill|
October 25, 2005
|Analysis of the Causes that Led to Eliza Jane Scovills Cardiac Arrest and Death|
What qualifies Dr. Al-Bayati to critique the analysis and conclusions of the LA coroner?
The required experience for a medical investigation such as the case of Eliza Janes death is a combination of Pathology (to analyze the medical data) and Toxicology (to investigate the influence of drugs, poisons and other chemicals). Al-Bayati is a pathologist and dual-board certified toxicologist. More details on his background are found below.
|Diplomate, American Board of Toxicology (DABT)|
| ||Al-Bayati was certified by the American Board of Toxicology in the areas of Human, experimental, regulatory and environmental toxicology).|
|University of California, Davis PhD in Comparative Pathology|
| ||Al-Bayati received a PhD in Comparative Pathology from UC Davis after studies including human pathology, medical immunology, medical biochemistry, analytical toxicology, environmental toxicology and experimental toxicology.|
|PhD Qualifying Exam|
| ||Al-Bayati entered the UC Davis PhD program after being examined by two MD pathologists from the universitys medical school. He passed on the first try.|
Some of Dr. Al-Bayatis most relevant publications are listed below. A complete list of his scientific publications since 1982 can be found on the National Library of Medicines PubMed website. Apart from the references below indicating his experience with unusual deaths of infants (often blamed on parents by doctors and police), Al-Bayati was the first researcher who reported pathologic changes in thymus and lymph nodes in experimental animals exposed to high levels of the pollutant Vanadium. He also discovered that the compound vanadate can cause renal fibrosis (kidney damage). This work is another clear indication of Al-Bayatis skill reading pathology slides in all organs and his evaluation of hematology data as well as his expertise in pathology and toxicology.
|2009||Analysis of causes that led to baby Huda Sharifs intracranial and retinal bleeding and fractures of the left humerus and the 7th|
|Medical Veritas. 6: 1977-91. |
Summary: Huda Sharif was 10weeksold when her parents took her to the hospital on July 13, 2008. She was diagnosed with subdural hygroma [a cyst], an acute left mid shaft humerus [upper arm bone] fracture, healed left 7th rib fracture, and minor retinal bleeding. Her blood coagulation and genetic osteogenesis imperfecta [weak bone] tests were normal. Her father was accused of causing the bone fractures and arrested. Huda and her 15monthold sister were placed with a foster family. Al-Bayatis investigation revealed that the likely source of the subdural hygroma was subdural bleeding. Huda was born at 40 weeks gestation by vacuum assisted Caesariansection and her head circumference increased by 2.1 cm within 5 days. It is likely that the vacuum instrument caused this bleeding. Increased intracranial pressure then led to retinal bleeding. Vitamin K, vitamin D 25hydroxy, and protein deficiency contributed to Hudas rib and humerus fractures. She had lost 167 g between July 7th and 24th. The synergistic actions among the following factors led to her humerus fracture: a) Vitamin and protein deficiency led to bone weakness; b) vaccines received on July 7th caused inflammation, edema, and the release of vitamin D, 1, 25dihydroxy. Hudas serum level of vitamin D, 1, 25dihydroxy was 166 pg/mL (369% of normal). It stimulated osteoclasts and increased bone resorption. Her length decreased by 1.9 cm within 6 days after vaccination; c) Edema led to her arm muscles to stretch and force the humerus to fracture at week points. Her weight gain rate after vaccination was 309% of her expected rate.
|Analysis of causes that led to Evyn Vaughns respiratory arrest, intracranial and retinal bleeding, and death|
|Medical Veritas. 6: 1938-59. |
Summary: Evyn, a 14monthold male, suffered from respiratory arrest on April 21, 2007, and was taken to Northwest Texas Hospital. He was treated with several medications but he did not improve. He was pronounced brain dead on April 22. Evyn was given heparin and his major organs were harvested for donations. At autopsy, the medical examiner (ME) observed intracranial bleeding, brain edema, widening of the sagittal suture, and optic nerve sheath hemorrhage. He alleged that Evyns bleeding, injuries, and death were caused by blunt trauma and his caretaker was accused of murder. Dr. Al-Bayatis investigation revealed that Evyn suffered from respiratory arrest as a result of bacterial infections, pneumonia, and septicemia. The likely causes of Evyns bleeding were infections and septicemias, liver damage, vitamin K deficiency, and heparin. Hypoxia, medications, bleeding, and increased intracranial pressure (ICP) caused his brain edema and the widening of the sagittal sature. The 27 vaccines given to Evyn and treatment with corticosteroids caused significant health problems and immune depression that led to his infections.
|Analysis of causes that led to baby Ron James Douglascardiopulmonary arrest, bleeding (intracranial, retinal, and pulmonary), and rib fracture|
|Medical Veritas. 6: 1960-77. |
Summary:Ron James Douglas was 16 weeks old when he choked after being fed and suffered a heart attack. Paramedics resuscitated him and took him to a hospital. He was pronounced dead at about 25 hours following admission. An autopsy was performed and the medical examiner (ME) found subdural, subarachnoid, and retinal bleeding and an acute rib fracture. The ME alleged that Rons death was caused by blunt trauma to the head and upper body. Rons father was accused of killing his son. Dr. Al-Bayatis investigation reveals that Ron suffered from a severe hypocalcemia which, in children, has caused seizures, respiratory disorders, laryngospasm, and/or severe cardiac disorders resulting in sudden death. It is the likely cause of Rons choking and cardiopulmonary arrest. He was born 11 weeks premature and developed hyperphosphatemia 39 days later. The likely causes of Rons hypocalcemia are hypoparathyroidism and hyperphosphatemia. He also developed hemolytic anemia and his thymus was involuted. Rons intracranial bleeding developed following admission to the hospital and the likely causes of his bleeding are acute liver injury, treatment with epinephrine, and disseminated intravascular coagulation (DIC). In addition, severe anemia contributed to his retinal bleeding. Cardiopulmonary resuscitation is the likely cause of Rons rib fracture.
|2008||Analysis of causes that led to baby Jackie Rays developmental delay and intracranial bleeding|
|Medical Veritas. 5: 1836-44. |
Abstract:A 3 monthold black male infant had a seizure and his mother took him to the hospital for examination. The baby was examined at the ER and no signs of trauma were identified. A CT scan exam of his head performed at 1.25 hours following his admission to the hospital (FAH) showed only a small area of bleeding in the left frontal region. A second CT head exam taken at about 40 hours FAH revealed that the baby had subdural hemorrhages and brain edema. A brain MRI exam performed at 89 hours FAH showed the babys subdural bleeding was bilateral and at various ages.
The treating physicians alleged that vigorous shaking (Shaken Baby Syndrome) caused the babys bleeding. The babys father was accused of causing his sons injuries. My investigation in this case reveals that the baby received 7 vaccines at two months of age, while he was ill. He suffered from developmental delay, anemia and femoral abnormalities. His head circumference (HC) was 38.7 cm on the day of vaccination and it decreased to 37.3 cm at 32 days post vaccination. The babys HC growth rate during the 2 months prior to vaccination was 2.8 cm/month.
His rate of weight gain was also reduced by 32% following vaccination as compared to the rate prior to vaccination. Vitamin K deficiency was the likely cause of the babys bleeding and femoral abnormalities. The allegations of Shaken Baby Syndrome and child abuse made in this case are false.
|Analysis of causes that led to toddler Steven Youngs respiratory arrest, intracranial and retinal bleeding, bronchopneumonia, peritonitis, and death|
|Medical Veritas. 5: 1775-1796. |
Abstract:Steven, a 25monthold white male, suffered from respiratory arrest and was resuscitated and taken to St. Josephs Hospital in Phoenix, Arizona. He was placed on mechanical ventilation and treated with epinephrine, IV fluids, antibiotics, ulcer prophylaxis, and sodium bicarbonate. No improvement in Stevens neurological condition was observed and he was pronounced dead 20 hours following admission.
An autopsy was performed and the medical examiner alleged that Steven died as a result of multiple blunt force injuries to the head and other regions of his body, and that the manner of death was homicide. Armando Castillo, Stevens caretaker was accused of killing Steven. He was arrested and indicted. A jury convicted him in May of 1999 and sentenced him to 27 years in prison for killing Steven.
My review of the medical evidence reveals that Steven was suffering from severe lymphocytopenia and immune depression, acute bronchopneumonia, bacterial infections, acute and chronic peritonitis, kidney infections, and liver damage. He had blood lymphocyte counts of 568693 cells/µL, which is well below normal. Gramstain study and blood culture of Stevens blood sample taken at 30 minutes following admission revealed the presence of Grampositive cocci and Staphalococcus coagulase negative.
Steven suffered from septic shock and vomited after he ate and the vomit blocked his airways and caused respiratory arrest. His intracrainal and retinal bleeding, other bleeding, and minor bruises were caused by infections, liver damage, and medications. The factual causes of Stevens illness, bleeding, and death were not revealed in court by the state and the jury convicted Armando based on a false theory.
|Analysis of causes that led to baby Ryans hemorrhagic pneumonia, cardiac arrest, intracranial bleeding, and retinal bleeding|
|Medical Veritas. 5: 1757-1774. |
Abstract:Ryan George was two months old when he suffered from cardiac arrest 9 days after receiving his second injection of Hepatitis B vaccine (HBV). He was successfully resuscitated at Coney Island Hospital in New York. His chest Xray and CT scan showed evidence of pulmonary edema, bleeding, and pneumonia. A CT exam of his head showed brain edema. He had a blood pH of 6.83 and a potassium level of 6.0 mEq/L. He was stabilized and transferred to Maimonides Medical Center (MMC).
Blood tests performed at MMC revealed that Ryan had an elevated band neutrophil count, hyperglycemia, hyperkalemia, hyperammonimia, hemolytic anemia, liver damage, hyperphosphatemia, and hypermagnesiumia. In addition, his PT, PTT, and INR were elevated. Ryan was treated with four types of antibiotics and other medications.
Ryan had an MRI head exam and an eye exam performed at 8 days and 9 days after admission, respectively. His MRI showed intracranial bleeding and his eye exam revealed retinal bleeding. Ryans father was accused of causing his sons injuries by shaking him vigorously (Shaken Baby Syndrome).
My investigation indicates that infection with Streptococcus pneumoniae caused Ryans illness and led to the development of hemorrhagic pneumonia, hemolytic uremic syndrome, kidney and liver problems, hepatic encephalopathy, seizure, coma, and cardiac arrest. The likely causes of Ryans intracranial and retinal bleeding are liver damage, infections, vitamin K deficiency, and severe anemia. Hepatitis B vaccine increased Ryan susceptibility to infection. The allegation of child abuse in this case is false.
|Analysis of causes that led to rib and skull fractures and intracranial bleeding in the case of the premature triplets Parneet, Sukhsaihaj, and Imaan|
|Medical Veritas. 5: 1589-1609. |
Abstract:Parneet, Sukhsaihaj, and Imaan were born at 30 weeks gestation via cesarean section and their weights were 1.31, 1.38, and 1.46 Kg, respectively. They suffered from respiratory acidosis, hypoglycemia, respiratory distress syndrome, sepsis, jaundice, gastroesophageal reflex, and anemia. They were discharged from the hospital at 42 days of age. The triplets were vaccinated with DTaP, IPV, Hib, and PCV vaccines at the age of 61 days in Imaans case and 75 days in Parneet and Sukhsaihajs case. At 15 days following vaccination, Parneet suffered from apnea, metabolic acidosis, seizures, infections, intracranial and retinal bleeding, and a skull fracture. Sukhsaihaj developed respiratory tract and eye infections, severe anemia, bleeding, and skull fracture. Imaan had severe anemia and a skull fracture. It was alleged that the babies injuries were caused by blunt trauma to the head. The parents were accused of causing the triplets injuries.
The clinical data described in this report reveal that the tripletÕs health problems were caused by vaccines. The severity of their injuries correlates with their hemoglobin levels at the time of vaccination. The subcutaneous and the intracranial bleeding resulted from protein and vitamin K deficiencies and infections. Severe anemia, vitamin K deficiency, and systemic infections are the likely causes of ParneetÕs retinal bleeding. The skull fractures resulted from protein and vitamin K deficiencies. The severity of the fractures correlates with the severity of the protein and vitamin K deficiencies, the degree of the infection, and the level of the intracranial pressure.
|Analysis of causes that led to rib and skull fractures, sudden illness, intracranial bleeding, and death in the case of toddler Roman Pitts|
|Medical Veritas. 5: 1573-88. |
Abstract: A male toddler suffered from cardiac arrest on May 26, 1988 after receiving amoxicillin. His stepfather, Daniel Childs, performed cardiopulmonary resuscitation (CPR) and called 911. The child was brought to the hospital, resuscitated, and given epinephrine 0.8 mg endotracheally and IV. He had a blood pH of 6.82, infection, and kidney and liver damage. The child was given sodium bicarbonate, antibiotics, and IV fluids. He died 4 days following admission.
An autopsy was performed and the medical examiner (ME) found separation of the coronal suture in the childs skull, a subarachnoid hemorrhage, brain edema, five healing rib fractures, and one acute rib fracture. It was alleged that the child died as a result of blunt trauma. However, the paramedics, physicians, and nurses at two hospitals did not observe any injury caused by trauma on the childs body. His X-rays and a CT scan of the head did not show skull fracture.
Daniel was accused of killing his stepson. He was convicted and sentenced to life in prison. The medical evidence presented in this report indicates that 1) the childs cardiac and respiratory arrest, pulmonary edema, and liver damage were caused by an allergic reaction to amoxicillin; 2) his subarachnoid bleeding was caused by epinephrine, liver injury, infection, and vitamin K deficiency; 3) the separation of the coronal suture was caused by the increased intracranial pressure which resulted from bleeding and edema; 4) the causes of the healing rib fractures were vitamin K and protein deficiency; 5) the acute rib fracture was caused by CPR; and 6) Daniel is innocent.
|2007||Analysis of causes that led to baby Stryker Eoghan Burkes sudden death (300KB pdf)|
|Medical Veritas. 4: 1470-83. |
Abstract: Stryker is a 55-day-old white male infant who was found dead in his bed on September 6, 2005. Based on the finding of methanol (20 mg/dL) and formic acid (23.2 mg/dL) in a blood sample taken from his heart at the time of autopsy, his parents were accused of poisoning him with methanol. My investigation reveals that the baby died as a result of severe hyponatremia. He had a critically low serum sodium level of 114 mmol/L (normal range: 135-145 mmol/L). Moreover, the gross and microscopic examinations of the brain showed evidence of edema and hypoxia and these lesions are reported in people suffering from hyponatremia.
My investigation also reveals that there is no evidence that the baby suffered from methanol poisoning. He did not show any symptom of metha-nol poisoning prior to his death. His brain, spinal cord, eyes and optic nerves, heart, lungs, liver, kidneys, pancreas, and other tissues were examined grossly and microscopically and no lesion was observed that indicated intoxication with methanol.
It is likely that the methanol and formic acid detected in Strykers blood resulted from the contamination of the blood with formalin used to fix tissues. This fixative contains 1 to 1.5% methanol and 3 to 4% formaldehyde. The oxidation of formaldehyde to formic acid is facilitated by formal-dehyde dehydrogenase present in the red blood cells.
|Analysis of causes that led to subdural bleeding, skull and rib fractures, and death in the case of baby Averial Buie (1.4MB pdf)|
|Medical Veritas. 4: 1452-69. |
Abstract: A female infant from Texas stopped breathing and her mother took her to the hospital. Blood analysis revealed the baby suffered from severe metabolic and respiratory acidosis, hyperglycemia, hyperkalemia, and lymphocytosis. A chest X-ray showed evidence of pneumonitis. Physical ex-amination revealed no evidence of injury caused by trauma. She was treated with epinephrine, sodium bicarbonate, antibiotic, and other medications. She developed bleeding outside the skull and intracranially. No skull or rib fractures were noted on the CT scans and X-rays taken during the fist four days following admission. However, skull and rib fractures were observed on the CT scan and X-rays taken at a later date.
Resuscitation efforts failed and the baby died at 11 days following admission. At autopsy, the medical examiner (ME) found healed skull and rib fractures, bleeding of various ages outside the skull and intracranially, and brain edema and necrosis. His microscopic examination of the H & E stained sections of the lung revealed evidence of bronchopneumonia, hyaline membranes in the alveoli, and bleeding. The ME alleged that the babys injuries were caused by trauma and her father was accused of killing her.
[Dr. Al-Bayatis] investigation reveals that the infant suffered from acute bronchopneumonia and respiratory distress syndrome on August 6, 2004, which led to hypoxemia, severe metabolic and respiratory acidosis, hyperkalemia, loss of consciousness, respiratory failure, and cardiac arrest. Her bleeding, brain edema and necrosis, and skull and rib fractures occurred in the hospital. These injures were caused by infection and medications
|A case of medically unjustified treatment with multiple mega doses of vitamin C with thyroid hormones that caused serious adverse reactions in a woman (302KB pdf)|
|Medical Veritas. 4:1235-43. |
Abstract: Christine, a 40-year-old white female, suffered suddenly from a fatigue syndrome in August 2000. Her blood and urine analyses, chest x-ray, abdominal ultrasound exam were normal. She consulted with several physicians who treated her symptoms and she made progressive recovery in her health. In February 2003, she felt that she had about 80% of her energy back and she was working full time. However, she consulted with a physician in California to get her full energy back and he recommended a detoxification treatment plan with high doses of vitamin C, glutathione, minerals, and vitamins. He gave her 39 intravenous injections of vitamin C (10-35 g per injection), glutathione (0.4-2.0 g per injection), calcium, and other vitamins for about 10 months. In addition, he also treated her with therapeutic doses of levothyroxine and cytomel for more than a year, although she had normal thyroid functions.
Christines clinical record indicates that she suffered from symptoms of hypercalcaemia and calciurea as a result of her treatment with vitamin C and calcium. The treatment with thyroid hormones also aggravated her condition and she became a 100% disabled. I have not found any medical justification for the use of detoxification agents or the treatment with thyroid hormones in this case. Christine’s blood, urine and hair analyses revealed that she was not exposed to chemicals at her workplace or home or ingested toxic chemicals. In addition, Christine’s blood thyroid hormones levels were within the normal range and she was not suffering from hypothyroidism.
|A missed case of poisoning with arsenic (215k pdf)|
|Medical Veritas. 4: 1244-50. |
Abstract: Christine, a 40-year-old white woman, suffered from acute gastrointestinal pain, diarrhea, malaise, and fatigue shortly after receiving oral herbal treatment and drank eight glasses of clear liquid in a clinic in California. On October 19, 2004 between 1540 and 1730, she was given Uro-well herbal supplement prescribed by her physician as a kidney-cleansing agent. Christine was transported by ambulance to the Stanford Emergency Department (SED) at approximately 1930. She was treated with activated charcoal orally and N-saline by IV. An electrocardiogram test showed that she developed sinus tachycardia. Christines blood test was negative for alcohol and her urine test was negative for the use of illicit drugs. The treating physicians did not order screening tests for the presence of heavy metals and arsenic in blood and urine, even though, she stated that a poison might be the cause of her symptoms.
Christine was released from SED after ten hours of admission. However, she continued to suffer from abdominal pain, fatigue, vomiting, and diarrhea for several weeks. A 24 hour-urine sample was collected and analyzed for arsenic on day 26 post-her hospitalization on October 19th. It revealed a significantly high level of arsenic (270 µg arsenic per 24 hour urine collection). Christines arsenic background level in urine was 18 µg arsenic per day. Furthermore, analysis of the Uro-well herbal supplement revealed arsenic level of 25 ppm, which is five-times the maximum permissible level of arsenic (5 ppm) in herbs set by the American National Institute of Standards and Technology. My investigation revealed that the exposure to a toxic level of arsenic by ingestion is the likely cause for Christines acute symptoms developed on October 19th.
|Analysis of causes that led to the development of vitiligo in Jeanetts case with recommendations for clinical tests and treatments (1.1M pdf)|
|Medical Veritas. 4: 1251-62. |
Abstract: At the age of two years, Jeanett developed vitiligo within days of receiving her first MMR vaccine and the fourth injection of DTaP and IPV vaccines. Furthermore, at five years of age, she developed many more unpigmented spots on her body with acrofacial vitiligo, following receipt of the second injection of MMR and the fifth injection of DTaP and IPV vaccines. Jeanetts susceptibility to developing adverse reactions to vaccine was notable a few hours after birth following receiving her first injection of the hepatitis B vaccine. Furthermore, the intensity and the frequency of her adverse reactions to vaccines were significantly increased following receipt of more doses of hepatitis B, DTaP, IPV, Hib, and MMR vaccines. Jeanetts health condition during her second year of life, when she was not given any vaccine was better than during her first year of life, when she received several vaccines.
It is likely that the MMR vaccine induced the depigmentation of Jeanetts skin through local and systemic autoimmune reactions. Synergistic actions between the MMR vaccine and other vaccines given to Jeanett could also be involved in causing the depigmentation of her skin. I believe that Jeanett should not receive any vaccines in the future. Vaccines probably will aggravate her present illness and trigger more illnesses. Jeanett was treated with corticosteroids ointment but the steroid did not help in stopping the depigmentation of her skin. Recommendations for clinical tests and treatment plans are presented in this report that I believe will help Jeanetts pediatrician to better monitor and treat her vitiligo.
|2006||Histopathological features of Eliza Jane Scovills and Destiny Jacobos lungs with analysis of the causes of death in both cases (pdf)|
|Medical Veritas. 3: 1041-8. |
Abstract: James K. Ribe is a supervising pathologist for the LA County Coroner who oversaw the investigations in Eliza Jane Scovills and Destiny Jacobos cases. Eliza Jane suffered from cardiac arrest and died following the administration of four doses of amoxicillin (400 mg twice a day). She died in Los Angeles, California on May 16, 2005 at the age of 3.5 years. Destiny Jacobo, a 21-month-old female toddler died suddenly in December of 1995 in Los Angeles. Ribe concluded that Eliza Jane suffered from Pneumocystis carinii pneumonia and died of AIDS. He listed the cause of death in Destinys case as shaken baby syndrome with associated head trauma. Ribe also alleged that there was forcible rectal insertion causing a retrorectal contusion.
My review of the medical evidence in Eliza Janes case clearly indicates that Eliza Jane died as a result of allergic reaction to amoxicillin and she did not die of AIDS. The histological features of her lungs show no evidence of inflammation and fibrosis. Ribes allegation that Eliza Jane suffered from Pneumocystis carinii pneumonia is not supported by the clinical data and medical facts. Furthermore, my review of the medical evidence in Destinys case revealed that she suffered from severe acute hemorrhagic pneumonia and septicemia. The bleeding in her case was caused by septicemia and vitamin K deficiency. It also shows that James Ribes allegations of shaken baby syndrome and sexual abuse made in Destinys case are not supported by medical facts.
|Analysis of causes that led to bleeding, cardiac arrest, and death in the case of Baby Nadine (pdf)|
|Medical Veritas. 3: 997-1012. |
Abstract: Ezbjörn Hahne was accused and convicted of killing his 40 day old daughter, Nadine, by shaking force (Shaking Baby Syndrome). Nadine suffered from cardiac arrest and died on September 16, 2004. The examination of her body and organs at autopsy and bone x-ray revealed no evidence of injuries caused by trauma. Ezbjörn was accused and convicted of killing Nadine based on the finding of old and new intracranial bleeding during autopsy.
My investigation of this case clearly indicates that Nadine died as a result of health problems and vitamin K deficiency that led to intracranial bleeding, edema of the brain, neurological problems, and cardiac arrest. Nadine was treated with three courses of antibiotics during her short life and had other predisposing factors for vitamin K deficiency. The evidence indicates that the intracranial bleeding occurred probably during the four weeks prior to Nadines death.
Nadine was born at 33 weeks of gestation by caesarean section. Her mother suffered from pregnancy complications that led to the premature rupture of the fetal membranes. She also had a urinary tract infection that was treated with antibiotics for 10 days following delivery. Nadine suffered from infection, hemolytic jaundice, neurological problems, and retardation of growth. She gained only 625 g during her 40 days of life (15.6 g/day), which is about 58% below of the weight gain expected for an infant her age.
|Analysis of causes that led to subdural bleeding and rib fractures in the case of Baby Patrick Gorman (pdf)|
|Medical Veritas. 3: 1019-1040. |
Abstract: Patrick and his twin sister, Peyton, were born 5 weeks premature. He suffered from acute abdominal and nonspecific symptoms at the age of 21&Mac218;2 months. CT scans, X-ray, and eye exams revealed that he had subdural and subretinal bleeding and seven rib fractures in various stages of healing. He also had severe anemia, thrombocytosis, low blood creatinine levels, hyperglycemia, and elevated neutrophills and monocyte counts. The treating physicians alleged that Patricks health problems resulted from shaking [shaken baby syndrome (SBS)] and child abuse. Patricks parents were accused of causing Patricks injuries.
My investigation revealed that Patricks acute symptoms resulted from acetaminophen intoxication. Patrick was treated with Tylenol/cold and he received about 200 mg of acetaminophen per day (64 mg/kg) and 3200 mg per 16 days. He was also treated with Zantac® (ranitidine) and Zantac® potentiates the hepatotoxicity of acetaminophen. The subdural and subretinal bleeding was caused by vitamin K deficiency, intoxication with acetaminophen, and severe anemia. The healed rib fractures occurred due to vitamin K and protein deficiencies and chronic coughing. It seems that the treating physicians alleged that Patricks health problems resulted from abuse, without considering the clinical data that lead to different causes, or performing differential diagnosis in this case.
|Comparative analysis of the autopsy reports of Destiny Jacobo and Eliza Jane Scovill (pdf)|
|Medical Veritas. 3: 767-71. |
Abstract: Destiny Jacobo, a 21 month-old Hispanic female toddler, died suddenly in December of 1995 in Los Angeles, California. The Los Angeles County Coroner, Dr. James K. Ribe, conducted an autopsy on Destiny on December 9, 1995 (Case No 95-09550). Ribe listed the cause of death in his report of February 13, 1996 as shaken baby syndrome with associated head trauma. He also alleged that there was forcible rectal insertion causing a retrorectal contusion. Destinys parents were accused of abusing and killing their daughter. They were convicted and sentenced to life in prison. The mother has been incarcerated since 1996; the father served five years of a life sentence before being released in 2001. Destinys family and their attorney requested that I review the autopsy report to identify the probable cause(s) of their daughters sudden acute illness and death. My analysis of the autopsy report indicates that the child died as a result of infections and vitamin K deficiency. It also shows that the allegation of sexual abuse is not supported by medical facts.
| ||Examining the causes of AIDS|
|Medical Veritas. 3: 901-13.|
|Analysis of the causes that led to Eliza Jane Scovills cardiac arrest and death.|
|Medical Veritas. 2 (2): 567-581.|
| ||Did Bryant Arroyo kill Baby Jordan Anthony Shenk, as alleged by the Commonwealth of Pennsylvania?|
|Medical Veritas. 2 (1): 367-82.|
|Assessment of adverse reactions to vaccines given to Greyson and Gwyneth with recommendations for clinical tests|
|Medical Veritas. 2 (1): 325-30.|
|Analysis of Causes That Led to the Bleedings in the Subdural Spaces and Other Tissues in Baby Alan Ream Yurkos Case|
|Medical Veritas. 1 (2): 231-8.|
|Analysis of Causes That Led to Baby Alan Ream Yurkos Cardiac Arrest and Death in November of 1997|
|Medical Veritas. 1 (2): 201-31.|
|Analysis of causes that led to Baby Roberts respiratory arrest and death in August of 2000|
|Medical Veritas. 1 (2): 179-200.|
|Shaken Baby Syndrome or medical malpractice?|
|Medical Veritas. 1 (2): 78-90.|
|Analysis of causes that led to Toddler Alexa Shearers cardiac arrest and death in November 1999|
|Medical Veritas. 1 (2): 86-117.|
|Analysis of Causes That Led to Baby Lucas Alejandro Mullenax-Mendezs Cardiac Arrest and Death in AugustSeptember of 2002|
|Medical Veritas. 1 (2): 45-63.|